avascular necrosis

Examples of avascular

avascular
We believe that our derivation of this simplified model indicates how models of avascular tumour growth arise as special cases of more physically based models.
As such, the paper provides a review of approaches currently being used to model avascular tumour growth.
This would be true even if the glucose concentration at the inner border of the avascular retina was assumed to be considerably lower.
The modified model does predict a steady-state stress distribution, and affords much insight into the mechanics of avascular tumour growth.
Therefore, many human visual problems may be due to failure of molecular mechanisms that generate the avascular zone.
This study shows that pit formation begins at the avascular zone during the late fetal quiescent period for retinal growth.
The epidermis is avascular and receives nutrients, regulatory mitogens and morphogens from the vascularized dermis.
In this paper a generalized model for the growth of avascular tumours is presented.
Many authors have proposed models of this form to describe avascular tumour growth, without referring to either the internal pressure field or the cell velocity.
Based on diagnosis, 58.6% of the patients had osteoarthritis and 3.1%, avascular necrosis.
Ideally, such a conclusion would be based on stress 0 strain measurements from vascular versus avascular primate retina.
In fact, the photoreceptor layer is perpetually avascular and is supplied by diffusion of materials from the choroidal circulation.
The model faithfully reproduces many of the features associated with avascular solid tumour growth such as evolution to a stable, structured steady state.
The dermis becomes thinner with age, more acellular and more avascular.
Since the cornea is an avascular tissue, hyperacute rejection, which results from vascular occlusion and is commonly seen in vascularised solid organ xenografts, is not seen in corneal xenografts.
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