functional deficit

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The extent to which a failure to support gamma-range oscillations is associated with other abnormal sensory-processing and functional deficits, however, is unknown.
Each technique-defined neuroscience could contribute to an understanding of the mechanisms underlying functional deficits observed in behavior.
At the moment there are two methods that show promise for future use in improving some of the functional deficits observed in antisocial behavior.
One assumption guiding interpretations from prospective studies is that the measurement of functional deficits is consistently valid from birth through childhood and into adulthood.
Extensive functional deficits do not, therefore, appear to be the result of large-scale death of autonomic neurons.
Further, these functional deficits in autism were associated with a structural defect, namely, decreased amygdala volumes.
Eventually, the developmental cascade would progress to the point at which functional deficits are manifest in cognitive performance and behaviors recognized as abnormal at home or at the clinic.
These should allow determination of the primary site of functional deficits, monitoring of progression and, ideally, detection of impairment of function before morphological changes are apparent.
As biological theories have become orthodox, hallucinations are more often thought of (by psychologists at least) as being caused by functional deficits in the brain.
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