Examples of apoptosis

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Apoptosis of tachyzoites was evaluated using light microscopy.
This means that neuronal apoptosis might be a correlation but not a cause of the gcm-robo1 doublemutant phenotype.
Neuronal death in amyotrophic lateral sclerosis is apoptosis: possible contribution of a programmed cell death mechanism.
The abrogation of tumor growth correlated with enhanced apoptosis in tumor tissues.
As opposed to necrosis (which refers to cell death by injury or insult), apoptosis does not lead to a reactive increase in neuroglial cells (gliosis).
The inhibition of apoptosis and promotion of growth are associated with clinical drug resistance.
The mechanism responsible for the destruction of the secretory epithelium is apoptosis, a form of programmed cell death that occurs in all multicellular animals.
Apoptosis is associated with the activation of caspases.
Those cells that are not induced to differentiate undergo apoptosis (programmed cell death).
Insufficient clearance of lymphocytes by apoptosis might be one reason why inflammatory responses become chronic.
The release of cytokines/chemokines indirectly contributes to apoptosis of bystander lymphocytes by upregulating a variety of genes with pro-apoptotic functions.
Under some circumstances, however, cytokines do influence whether a cell undergoes apoptosis.
The occurrence of apoptosis in the embryo can be decreased by adding growth factors to the culture media.
Thus, after the mid-gastrula stage, the embryo responds to nocodazle by undergoing apoptosis.
The apoptosis cascade is the best described sequence of events during turnover of villous trophoblast.
These agents have been directed towards glutamate homeostasis, apoptosis, oxidative stress and neuroprotection.
Furthermore, normal features of cell death in the form of coordinated apoptosis could be thrown into disarray by damaging feedback influences from degradation products.

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