Examples of emesis

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Reduction of cisplatininduced emesis by a selective neurokinin-1-receptor antagonist.
Acute emesis usually begins 2 to 4 hours after cisplatin administration and peaks at 6 hours (23;26).
The role of serotonin blockade in the pathophysiology of chemotherapy-induced emesis is wellestablished clinically through the serotonin receptor antagonists, which include ondansetron, granisetron, dolasetron, and tropisetron (14;28).
Ondansetron versus granisetron, both combined with dexamethasone, in the prevention of cisplatin-induced emesis.
The data relied on a small randomized trial that did not report the number of emetic episodes in patients who had emesis nor the type of rescue therapy administered.
These rates of complete emesis control were subsequently incorporated into the decision-analysis model.
Therefore, costs related to breakthrough emesis were not entered in the baseline analysis.
Research efforts to prevent delayed emesis have been directed at blocking neurotransmitter receptors in the brainstem's vomiting center.
Delayed emesis can persist up to five days after cisplatin administration, peaking in intensity at 48 to 72 hours.
Patients require pre-treatment with the extra-cerebral dopamine receptor antagonist domperidone but may still experience severe emesis.
He had heard about the episode of emesis.
For this reason, cisplatin-induced emesis has become the human model by which antiemetic regimens are evaluated (7).
Previous studies have established that patients who have complete control of acute emesis are less likely to experience delayed emesis (26).
The therapeutic area selected as the case study was a new antiemetic agent for the prevention of chemotherapy-induced emesis.
Therefore, there is a need to identify new agents that will improve the control of delayed emesis.
Other sensitivity maneuvers consisted of adding the cost of emesis and secondary therapy for breakthrough vomiting to the model.
Emesis control rates for this and other scenarios were also obtained from published randomized trials (11-13; 17-20).
The rates of complete emesis control were abstracted from a double-blind randomized trial.
The severity and pattern of chemotherapy-induced emesis depends on the chosen drug regimen, dose, route, and schedule of administration (23;29;32).

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