Examples of cholinesterase

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Additional tr ials of cholinesterase inhibitors enrolling patients with high levels of neuropsychiatr ic sy mptoms may be warranted.
Davtyan (1977) observed an increase of the redox processes and the activity of cholinesterases and hyaluronidases in the host with helminthiases.
Use of cholinesterase inhibitors is associated with an increased risk of new-onset or worsening incontinence.
Peptidergic immunoreactivity was apparent in both the central and peripheral nervous systems and the general pattern of staining approximated that observed for cholinesterase activity.
Tacrine is a reversible, noncompetitive cholinesterase inhibitor with a short half-life of 1.4-3.6 hours.
What are the effects of the newer cholinesterase inhibitors, and do they exaggerate bradycardic responses?
At baseline, the two groups were not significantly different with respect to demographic characteristics, disease severity, or percentage of persons taking cholinesterase inhibitors.
The use of tacrine, and the more recently released cholinesterase inhibitors, will be discussed in detail below in the following section.
Next, we describe a rational framework for understanding why medications such as cholinesterase inhibitors and other agents currently in clinical trials, might be effective.
Prescribing anticholinergic drugs to counter this adverse effect can reduce any cognitive benefit from cholinesterase inhibitor treatment.
Two classes of anti-dementia agents have emerged in the recent past: anti-oxidants and cholinesterase inhibitors.
Several approaches have been tried including precursor loading, cholinesterase inhibition and direct and indirect cholinergic receptor stimulation.
Both calcium antagonist (especially nimodipine)189 and the newly-introduced cholinesterase inhibitors190 have also shown potential in treating depression in dementia patients.
Further investigation of the use of cholinesterase inhibitors in traumatic brain injury is needed.
The cholinesterase and acetylcholine content of the chick retina with special reference to functional activity as indicated by the pupillary constrictor reflex.
Rivastigmine is a potent inhibitor of cholinesterases in plaques and tangles.
Eserine controls for cholinesterase activity were at all times negative.
Most of the assessment scales used in the assessment of cholinesterase inhibitors do not specifically rate sleep as an end-point but only as part of the multidimensional behaviour assessment.

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