Examples of verapamil

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Catheterization at 5.5 months of age revealed a non-obstructive form of hypertrophic cardiomyopathy with elevated left ventricular end diastolic pressures that regressed with verapamil.
The decision was made to continue treatment with verapamil because of the improved clinical status.
Under the influence of verapamil, the dynamics of replenishment of intracellular calcium deposits were significantly changed.
The other subject received intravenous verapamil in the intensive care unit in an acute setting of labile blood pressure and severe congestive heart failure.
Ability to complete meiotic maturation was markedly diminished in oocytes cultured in medium supplemented with verapamil.
Chronic treatment with verapamil continued in the group with primary cardiomyopathy.
It is our feeling that careful, long-term monitoring for atrioventricular nodal dysfunction is needed in all infants on verapamil.
After treatment with verapamil, the heart rate decreased to 150-220 bpm.
The replenishment of calcium deposits is significantly changed under the effect of verapamil.
Given our intention to treat, the initial response to verapamil given acutely in all cases is therefore reported.
Oral verapamil at 3-5 mg/kg/day was started after catheterization.
The role of verapamil in this death is unclear though concerning given the finding of complete heart block.
In no instance did infusion of verapamil result in worsening pulmonary oedema or an increase in respiratory distress.
Both enantiomers cause vasodilatation and lower blood pressure while l-verapamil is a more potent cause of heart block and heart failure.
Following cardiac catheterization, oral verapamil was started at 3-5 mg/kg/day.
There was one death in the two patients with primary hypertrophic cardiomyopathy who were not treated orally with verapamil.
We evaluated prospectively the safety and efficacy of verapamil in infants.
The second patient on long-term verapamil treatment died suddenly after 37 months of follow-up.

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