Examples of thromboembolism

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Even if asymptomatic thrombosis is excluded however, the incidence of symptomatic thrombosis and thromboembolism was high, at 5%.
The mean age at surgery in patients with thromboembolism was 4 years compared to 4.8 years in the overall group, the difference is not significant.
In none of the patients did we observe arrhythmias, thromboembolism, endocarditis, death, morbidity or other disorders.
With mitral stenosis there is an increased risk of atrial fibrillation, thromboembolism and pulmonary oedema.
The challenge is to ensure implementation of effective prophylactic strategies in appropriate patient groups who are at risk for venous thromboembolism.
How can venous thromboembolism best be treated, and for how long?
Osteoporotic fractures and the recurrence of thromboembolism during pregnancy and the puerperium in 184 women undergoing thromboprophylaxis with heparin.
For patients with a persistent dilated coronary artery after surgery, we recommend some form of anti-platelet therapy to prevent possible thromboembolism involving the affected artery.
Inherited thrombophilia and first venous thromboembolism during pregnancy and puerperium.
There were no incidences of postoperative thromboembolism or endocarditis.
While initially salutory, persistence of the right-to-left shunt may not be desirable due to presence of chronic cyanosis and risk of thromboembolism.
How great is the problem of venous thromboembolism?
Cause of late death included sudden dysrhythmia, univentricular failure, thromboembolism, protein-losing enteropathy, and accidental.
Extended bed rest may cause complications such as joint stiffness, muscle atrophy, osteoporosis, pressure sores, and thromboembolism.
The primary endpoint was a composite of the rate of thromboembolism, severe hemorrhage and valve-related mortality.
Prevention and treatment of venous thromboembolism in older people ongoing risk factors need long-term anticoagulation.
There have been no hospital deaths or documented thromboembolisms to date.
There is, however, some reason to suppose that the risk of thromboembolism is dose-dependent and largely attributable to the oestrogen moiety.

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