Examples of synaptogenesis

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Thus, it appears that learning, not merely neural activity, is required to induce synaptogenesis.
For example, in the human, migration is virtually complete at birth and synaptogenesis is in progress.
First, our understanding of the cellular and molecular mechanisms that underlie neurotrophins and other cell-cell signals that modulate synaptogenesis is in its infancy.
Experience-independent synaptogenesis refers to the formation of synapses without requirement for experiential input; an example is the development of taste buds on the tongue.
The teleost optic nerve has been the focus of numerous studies investigating regeneration and synaptogenesis.
Ethanol-induced apoptosis in the developing visual system during synaptogenesis.
Using this measure, it can be shown quite straightforwardly that a system can acquire more powerful conceptual structures as activity-dependent synaptogenesis constructs more powerful representations.
However, differentiated neurons do not exhibit spontaneous synaptic current, providing an opportunity to identify the crucial steps and signaling molecules that regulate synaptogenesis.
This reduction was a product of synaptogenesis and not habituation, because the tilt persisted in animals with permanent retinal damage.
Postnatal synaptogenesis and neuronal connectivity depend on the maturation of a number of cell types including astroglial cells.
Importantly, in the last decade, it has been shown that astrocyte-derived signals are necessary for synaptogenesis.
In the second half of gestation there is a brain growth spurt with axonal and dendritic growth, enhanced synaptogenesis, myelination and neuroglial proliferation.
Furthermore, it appears that this interaction prevents the triggering of axonal outgrowth and synaptogenesis in the adult cerebellum.
Alterations in other calcium-dependent mechanisms such as cell attachment, migration, processes elongation, and synaptogenesis could reasonably account for the disruption observed.

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