Examples of perivascular

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Moreover, the immune response characterised by perivascular inflammation induces further lowgrade damage to the vascular endothelium.
Abnormalities such as perivascular brightness, which is an increased echogenicity of the walls of the coronary artery, and transient dilatation of the coronary artery, can also be noted.
As shown by the histological sections, persistent inflammation is associated with an abnormal increase in the number and distribution of leukocytes within the tissue, classically as a perivascular infiltrate (fig001cbb).
Histopathological examination in four cases demonstrated a perivascular lymphohistiocytic in®ltrate with occasional eosinophils, with both upper dermal oedema and focal epidermal oedema causing areas of vesicular spongiosis.
T cells out of the perivascular space, which continue to associate with astrocytes via interactions of adhesion molecules.
Astrocytes in the infected mice appeared to display increased affinity for the perivascular region.
Calcifications generally develop from vessel walls and the perivascular space, and progressively extend towards neurons, which could remain undamaged for a long time.
Histological changes observed included perivascular infiltration with eosinophils and mononuclear cells, peribronchiolitis, alveolitis and mucus production.
Pulmonary arteries showed thickening of the media and perivascular fibrosis.
These are probably released from infected and/or activated perivascular macrophages and resident microglial cells.
A basement membrane, which includes embedded pericytes, together with the astrocyte foot processes provide the structural basis for the perivascular space.
A moderate, mainly perivascular inflammatory response and large inflammatory foci with a strong alveolitis were present in lungs of both strains of mice.
There is little perivascular fat, and the artery is almost suspended on a mesentery.
Focuses were perivascular in eight cases out of 24.
The inflammation extended into the perivascular epicardial fat and adjacent myocardium.
Another striking and unexpected finding in the present study was perivascular inflammation associated most prominently with those lobes subjected to pulmonary venous banding.
Clusters of lymphocytes and plasma cells, many of which were perivascular in distribution were seen in areas remote from obvious necrotic foci.

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