Examples of neuroprotective

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The neuroprotective agent riluzole inhibits release of glutamate and aspar tate from slices of hippocampal area.
The potential antioxidant and neuroprotective properties are offset by the theoretical risk of cerebrovascular ischaemia.
The objectives of the current study were to further investigate and elucidate neuroprotective mechanisms of minocycline under a variety of conditions.
The first area is the exact mechanism and pathways by which lithium exerts its neuroprotective effects.
First, in bipolar disorder lithium's neuroprotective properties imply a new aetiology and hence treatment paradigm.
These findings have important implications in the search for effective neuroprotective therapies in treating stroke.
The consequences of the definitively establishing such a neuroprotective effect would be considerable.
They have also been shown to be neuroprotective to retinal cells in a variety of injury models.
In our study brimonidine had a neuroprotective effect in the retina of rats with chronic hypertension.
However, recent data suggest that at least some of the complement components have the ability to contribute to neuroprotective pathways.
Both of these pathways are strongly associated with neuroprotective effects.
From the lab to the clinic: activation of an alpha-2 agonist pathway is neuroprotective in models of retinal and optic nerve injury.
In addition to reducing production of inflammatory mediators, it has been postulated that minocycline might also be directly neuroprotective under these circumstances.
In conclusion, we believe that brimonidine has dual neuroprotective effects in the chronic ocular hypertensive rat retina.
Second, lithium's neuroprotective properties suggest that lithium might have use in preventing or treating brain damage after an insult has occurred.
This mechanism may exert a neuroprotective effect and this is currently under clinical investigation.
Lithium has also proven to be neuroprotective in vivo.
In this case, since lithium is neuroprotective, it is possible that neuroprotection is important to the treatment of bipolar disorder.
This was a promising beginning for a potential neuroprotective compound although further studies failed to establish a drug-placebo difference.
We studied candidate genes that could mediate this neuroprotective effect.

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