Examples of neurodegeneration

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Interactions between neurons and microglia play a central role in the balance neurodegeneration/neuroprotection.
They include genes in which mutations cause abnormal ion-channel function, disordered brain development, progressive neurodegeneration and disturbances of cerebral energy metabolism.
We also found that administration of glutamate pyruvate transaminase (alanine aminotransaminase) with pyruvate diminished the neurodegeneration during simulated ischemia.
We have not observed inflammatory cells in the retina of any eyes either during the neurodegeneration or at other times.
Protofibrils, pores, fibrils, and neurodegeneration: separating the responsible protein aggregates from the innocent bystanders.
How polyglutamine induces neurodegeneration, and why neurodegeneration occurs in only select neuronal populations, remains a matter of intense investigation.
A -clearing capabilities and become more pro-inflammatory, therefore contributing to neurodegeneration.
Protofibrils, pores, fibrils, and neurodegeneration : separating the responsible protein aggregates from the innocent bystanders.
A combination of these therapeutic approaches with anti-inflammatory drugs such as indomethacin might be of clinical benefit in the treatment of neurodegeneration.
Cellular factors responsible for initiating these events and the mechanism by which they contribute to the neurodegeneration and cytotoxicity remain poorly understood.
These have been illustrated by commenting on recently described studies in the realm of depression and neurodegeneration.
Interactions between wild-type and mutant prion proteins modulate neurodegeneration in transgenic mice.
These neuropathologies are both believed to lead to neurodegeneration.
Reactive microcytes, proposed to prevent damage within their first 3 days of reactivity, act mainly as inflammation amplifiers and mediate neurodegeneration.
Serial transmission in rodents of neurodegeneration from transgenic mice expressing mutant prion protein.

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