Examples of microglia

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Thus, there is a parallel relationship between the density of microglia/brain macrophages increase and demyelination progression.
Microglia can be present in functionally and morphologically different activation states, including resting (ramified), activated (hypertrophied), and phagocytic microglia.
These activated spinal microglia are mainly residential microglia, but might also include migrating microglia from the circulation.
After a few minutes axonal fibers generated bulbous endings which were partly engulfed by microglia and removed.
Because this cannot be determined by examining morphology alone, the data described below provide additional experimental evidence to support a functional change in microglia.
Interactions between neurons and microglia play a central role in the balance neurodegeneration/neuroprotection.
Additionally, studies have shown that microglia in vivo experience an age-related decrease in telomere length.
In this review, we focus on how activation of microglia controls abnormal pain.
Plaques contain variable numbers of activated microglia as well as reactive astrocytes surrounding the core.
This enables us to characterize the functional receptors and the physiological phenotype of microglia in situ.
Alternatively, stimulated microglia undergo senescence, demonstrated by reduced viability and mitotic activity, after several weeks in culture and in the absence of telomerase repression.
The nerve injury-induced spinal cord microglia and astrocyte activation is reduced in the toll-like receptor 2 knockout mice.
Consequently, there is an ongoing need to parallel studies of rodent microglia with investigation of their human counterparts.
Microglia have been implicated in aging-related neurodegenerative disease, yet relatively little is known about how normal aging and chronic pathological states may affect microglial function.
Microglia are innate immune cells and form the first line of defence of the central nervous system.
We hypothesized that microglia activated under various conditions may be functionally different.
Microglia are resident immune cells in the brain that become activated in response to diverse central nervous system injuries.
Both microglia and astrocytes are activated in these pain models and interact with neurons in the complex pain pathophysiology.

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