Examples of glutamatergic

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By immunogold-electron microscopy, we demonstrated that both receptors are highly express and colocalized at the synaptic glutamatergic membranes and surrounding astroglial membranes.
The correlation of these both neurochemical parameters with the neuroprotective actions of guanosine against glutamatergic neurotoxoicity has to be elucidated.
This mechanism is likely to affect glutamatergic inputs differentially according to their firing rate.
We speculate that astrocytes may serve as integrators, linking serotonergic and glutamatergic pathways.
The neurexins interact with the neuroligins, and together they are important for glutamatergic synaptogenesis.
Glial regulation of synaptic plasticity at glutamatergic synapses is the subject of two papers in this issue.
Clinically, such a cortico-cortical spread of glutamatergic-mediated overexcitation may be reflected in the occurrence of catatonic syndrome in schizophrenia.
Likewise, glutamatergic mediation of all memory and cognitive processes in all mammals, makes "higher social brain" assumptions unparsimonious.
Characterizing their glutamatergic responses with whole-cell recordings ensured the physiological identity of these cells.
We have now demonstrated that prenatal inflammation suffices to induce deferred glutamatergic synaptic dysfunction in adult.
Subtle alterations in glutamatergic receptors underlie the aging-related decline in hippocampal function.
Furthermore, thalamic inputs can also gain access to basal ganglia output nuclei via subthalamo-pallidal projecting neurons, neurons receiving glutamatergic thalamo-subthalamic projections.
It will be important to continue to seek bridges between glutamatergic and neurodevelopmental models of schizophrenia as these two parallel theoretical avenues advance.
Given these findings, further research with medications that specifically affect the glutamatergic system is warranted.
Due to glutamatergic-mediated connections from medial temporal areas, activity in lateral orbitofrontal/prefrontal cortex may be deregulated in schizophrenia.
For example, not all neuropathological changes in schizophrenia are limited to glutamatergic pyramidal neurons in prefrontal cortex.
It has been increasingly clear that the glutamatergic system is involved in plasticity or "synaptic modifiability," an inherent property of neurons during maturation.
However, based on available evidence, its therapeutic effect may result from a reduction of excitatory, glutamatergic neurotransmission in brain networks regulating emotional expression.

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