Examples of autoimmunity

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A later age of onset is normal for this clinical subclass and it is associated more often with autoimmunity.
We discuss the evidence that cytokines can either promote or protect against autoimmunity and review the use of individual cytokines as therapeutic agents.
By doing this, the contribution of infective agents to pathogenic processes such as autoimmunity and vascular disease can be truly evaluated.
Furthermore, restoration of homeostasis may be difficult to achieve because insufficient or excessive cell activation can predispose to autoimmunity.
Several microbial antigens have been identified as potentially important in the process of the induction of autoimmunity by molecular mimicry.
Another example of extremely harmful immune responses is autoimmunity, where the immune system attacks the individual's own tissue.
Hence, it has been hypothesised that helminths may protect against the development of autoimmunity and allergy.
A defect in these functions can give rise to autoimmunity.
Tolerance is the process that eliminates or neutralises autoreactive cells; thus, a dysfunction in this system can cause autoimmunity.
Both immunodeficiency and exaggerated immune responses can predispose to autoimmunity.
Evidence is also unsubstantiated as yet for alternative aetiological hypotheses such as autoimmunity, atopy, and genetic predisposition.
In other words, autoimmunity is a normal characteristic of the immune system, which constantly seeks to identify and monitor key host constituents.
The ultimate goal is to selectively correct deficits that contribute to autoimmunity without impacting protective immunity.
The nature and mode of action of nematode-derived molecules with immunomodulatory properties are considered, and their therapeutic efficacy in models of autoimmunity and allergy described.
Subsequently, the simplicity of this polarised response has been challenged, and it is now clear that t h e phenotypes involved in tolerance and autoimmunity are not absolute.
Mannose binding lectin polymorphisms are associated with early age of disease onset and autoimmunity in common variable immunodeficiency.
Development of symptoms or signs of autoimmunity have not been reported.
Chagas' disease aetiology : autoimmunity or parasite persistence?

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